Grade C · Emerging No Human Data Glu-Asp-Arg (tripeptide)

Pinealon

Also known as: Pinealon · EDR · Glu-Asp-Arg

NOT MEDICAL ADVICE · NOT FDA-APPROVED. This page summarizes what has been published about Pinealon in the research literature. It is not a protocol, not a dosing recommendation, and not an endorsement. Pinealon is not FDA-approved for human use and is not legally compoundable in the United States. Do not self-administer. Consult a licensed healthcare provider.

Research focus

Neuroprotection in cognitive aging models; oxidative-stress resistance; geroprotection.

US regulatory status

Not FDA-approved · Not compoundable

Evidence rating

No Human Data

Origin

Designed by the Khavinson group as a synthetic tripeptide modeled on fragments identified in pineal extract research. Developed primarily for experimental neuroprotection and anti-aging studies.

Plain-language summary

Pinealon is a three-amino-acid synthetic peptide designed as a research tool in Khavinson-group geroscience work. Available data is largely from cell culture and rodent studies suggesting cognitive and antioxidative effects. Human data is very limited. It is not FDA-approved and not legally compoundable in the United States.

Claimed mechanism (as reported)

Russian literature proposes that Pinealon penetrates the blood-brain barrier, influences gene expression in neural tissue, and reduces markers of oxidative injury in cultured neurons and animal brain. Mechanism details remain at the preclinical-proposal stage; the neuron-targeting model has not been independently validated outside the originating research group.

Evidence summary

Preclinical only. Cell-culture and rodent behavioral studies form the bulk of the literature. English-language publications are scarce and concentrated in a small number of authors. No published randomized controlled trials in humans as of 2026.

What the research reports

Neuroprotective effect of Pinealon on neurons under oxidative stress (preclinical)

Grade B

Arutjunyan A, Kozina L, Khavinson VKh et al. · Bulletin of Experimental Biology and Medicine · 2012–2017

Reported finding: Reported reduction in oxidative-stress markers and improved survival of cultured neurons exposed to glutamate or hypoxia. Rodent behavioral assays (Morris water maze variants) showed performance improvements in aged or prenatally-stressed animals.

Sample: In-vitro and rodent models only

Methodology: B (preclinical) — reproducible cell-culture findings within the originating group

Limitations: No independent replication outside the originating group; no human data.

PubMed →

Administration reported in studies

Published animal studies use intranasal or intraperitoneal dosing in microgram-per-kg ranges. No human dosing data exists in the peer-reviewed literature. This section describes research conditions only.

This section reports what published studies describe. It is not a dosing recommendation from TPS.

Safety record

No human safety data available. Preclinical tolerability in rodent models appears acceptable within the limited published follow-up. Long-term or reproductive safety data does not exist.

US legal status

Not FDA-approved. Not on the 503A bulk list. Not legally compoundable in the United States. Internet-sourced 'research chemical' material is not verified for purity or potency by TPS.

Open research questions

  • ? Does Pinealon cross the blood-brain barrier meaningfully in humans at any dosing route?
  • ? Are the antioxidative cell-culture findings reproducible in independent laboratories?
  • ? What human pharmacokinetic and safety data would be required before a Phase 1 study?

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Summaries of the Russian and English-language literature, bias-checked and plainly framed.

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